Research Suggests mRNA COVID Vaccines Produce Spike Protein in Heart Cells – but the Anomalies Are Different

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Recent research conducted in Germany has unveiled significant insights into the effects of COVID-19 mRNA vaccines on heart cells in both rats and humans. Within a short span of 48 hours after vaccination, these vaccines prompt the formation of spike proteins.

These proteins, generated from the mRNA instructions within the vaccines, were identified within the heart cells. Interestingly, both Pfizer and Moderna vaccines resulted in cell irregularities, albeit of different kinds.

Dr. Peter McCullough, a distinguished internist, cardiologist, and epidemiologist, who has authored numerous research reports and pioneered widely employed treatment approaches for SARS-CoV-2 patients, notes that this rapid manifestation of cellular changes indicates a potential mRNA toxicity reaction. The rapidity of this transformation within just 48 hours is a notable finding.

The research posits crucial implications for the diagnosis and treatment of cardiac events post mRNA-based COVID vaccination. Furthermore, these findings may elucidate enduring cardiac symptoms in individuals grappling with long-COVID.

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In this study, mRNA vaccinations were introduced to cell cultures originating from rat and human heart cells. Notably, Moderna and Pfizer vaccines encompass distinct mRNA doses, with Moderna presenting a higher dosage of 100 micrograms in contrast to Pfizer’s 30 micrograms. Consequently, both groups were administered 100 micrograms of mRNA.

Within a mere 48 hours, researchers were able to discern the presence of spike proteins in both sets of cell cultures, along with the observation of aberrations in heart contractions. The supplementary video supplement further illustrated the contrast between regular contractions in an unvaccinated rat heart cell and those in vaccinated cells.

The response from Pfizer-vaccinated cells was marked by more robust and sustained contractions, attributed to heightened protein kinase A (PKA) activity, a factor directly tied to heart performance. On the other hand, Moderna-vaccinated cells exhibited irregular contractions and disrupted calcium regulation, influenced by disturbances in RyR2 receptors crucial to coordinating heart contractions.

Additionally, some heart muscles treated with Moderna vaccines displayed irregular and peristaltic contractions, while others exhibited irregular and arrhythmic contractions. The presence of spike proteins was also confirmed within 48 hours in the cell culture of human cells.

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At the cellular level, the impacts of the COVID-19 vaccines seemed to align more closely with cardiomyopathy than with myocarditis. Unlike the latter conditions, where heart muscles become inflamed and damaged, cardiomyopathy entails structural and functional abnormalities in the absence of other heart diseases.

Notably, prior research by Dr. James Gill suggested that anomalies following COVID-19 Pfizer vaccination may resemble cardiomyopathy induced by toxic stress rather than classic myocarditis.

While the research primarily focused on animal cells, it strongly implies potential vaccine cardiotoxicity. The identified heart irregularities, likely stemming from disruptions to RyR2 and elevated PKA protein levels, represent significant risk factors for sudden cardiac death and related complications.

Dr. McCullough highlights the distinctive responses to the two vaccines, hinting at underlying cell toxicity related to the mRNA codes. This is a significant finding, as it raises concerns about potential cardiac toxicity in other mRNA vaccines in development.

This study is part of a growing body of research shedding light on the cardiotoxic effects of mRNA vaccines. A Japanese study demonstrated metabolic shifts in vaccinated individuals, suggesting abnormal heart function.

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Similarly, a Harvard study found traces of COVID-19 mRNA in the hearts of those who passed away within 30 days of vaccination. These studies collectively contribute to the ongoing discourse surrounding mRNA vaccine safety and its implications for cardiac health.

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